Volitional control of facial expression
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Primate Communication And Human Language
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In social situations facial expressions are often strategically employed. Despite the many research on motor control of limb movements, little is known about the control over facial expressions. Again, peripheral lesions make facial paresis occur, and two types of facial paresis that we learned about in class are volitional and emotional. Bell's palsy is the most common type of lesion that causes facial paresis (Larner, ). I was interested in volitional and emotional facial paresis after our class discussion on it.
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Facial expressions reflect decisions about the perceived meaning of social stimuli and the expected socio-emotional outcome of responding (or not) with a reciprocating expression. The decision to produce a facial expression emerges from the joint activity of a network of structures that include the. If the brain has two means of producing facial expressions—a deliberate and a nondeliberate or "emotional" means—one might expect to find a disorder in which patients can make facial expressions in response to verbal instructions but not in response to the patients' own emotional states.
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The interesting thing about volitional facial paresis is that the patient cannot voluntarily move the facial muscles but will express a genuine emotion with those muscles. In contrast, emotional facial paresis is caused by damage to the insular region of the prefrontal cortex, to the white matter of the frontal lobe, or to parts of the thalamus. substrate for facial expression. There are two distinct neural pathways that mediate facial expressions, each one originating in a different area of the brain. Volitional facial movements originate in the cortical motor strip, whereas it has been suggested that spontaneous facial expressions originate in the subcortical areas of.
Volitional control of anterior insula activity modulates the
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control their facial activity in the presence of a humorous stimulus (which in a dual factor theory provides a push to facial activity), limits of volitional control have been demonstrated. Facial inhibition tasks have been used previously, particularly in the con-text of facial feedback studies (e.g., Bush, Barr, McHugo, & Lanzetta, ;.
Impairs ability to recognize facial expressions of emotion Volitional facial paresis Difficult to move face muscles voluntarily due to primary motor cortex damage-can express genuine emotion w/ these muscles.
The Production of Emotional Expressions is not Generated in Motor Cortex Volitional Facial Paresis following damage to face region of motor cortex Cannot voluntarily move facial muscles on affected side of face but show normal involuntary facial expression of emotion Emotional Facial Paresis following damage to insular region of prefrontal cortex Show no emotion on affected side of face but.
In addition, it has been noted that many facial expressions shown by primates bear striking similarities to human expressions. This has lead to the hypothesis that facial expression might be genetically determined. Facial expressions are produced by the synergistic or co-operative action of many different facial muscles.
Methods: 12 persons with stable schizophrenia and matched healthy controls underwent a standardized procedure for posed and evoked facial expressions of five universal emotions, including happy, sad, anger, fear, and disgust expressions, at three intensity levels. Subjects completed self-ratings of their emotion experience.
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